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Pharmacological investigation of spreading depression propagation in rat neocortical tissues

von Aziza El Harrak

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[1.] Aeh/Fragment 020 01 - Diskussion
Zuletzt bearbeitet: 2014-04-29 22:26:29 Schumann
Aeh, Fragment, Gesichtet, KomplettPlagiat, SMWFragment, Schutzlevel sysop, Sheikh 2009

Typus
KomplettPlagiat
Bearbeiter
Hindemith
Gesichtet
Yes
Untersuchte Arbeit:
Seite: 20, Zeilen: 1-9
Quelle: Sheikh 2009
Seite(n): 24, Zeilen: 5-13
GABAergic inhibitory networks can also synchronize principal cells in the neocortex and hippocampus (Cobb et al. 1995). Higashima et al. (1996) have shown that activation of GABAergic mechanisms is necessary for the generation of afterdischarges recorded in hippocampal slices after electrical stimuli. Experimental and computational data obtained by Traub et al. (1996) also suggest a role played by GABAA-mediated depolarizing conductance in the epileptiform synchronization that occurs in some models of epileptiform discharge (in particular that induced by 4AP application). GABAergic inhibitory networks can also synchronize principal cells in the neocortex and hippocampus (Cobb et al. 1995). GABAergic inhibitory networks can also synchronize principal cells in the neocortex and hippocampus (Cobb et al. 1995). Higashima et al. (1996) have shown that activation of GABAergic mechanisms is necessary for the generation of afterdischarges recorded in hippocampal slices after electrical stimuli. Experimental and computational data obtained by Traub et al. (1996) also suggest a role played by GABAA-mediated depolarizing conductance in the epileptiform synchronization that occurs in some models of epileptiform discharge (in particular that induced by 4AP application). GABAergic inhibitory networks can also synchronize principal cells in the neocortex and hippocampus (Cobb et al. 1995).
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Sichter
(Hindemith) Schumann


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