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[1.] Br/Fragment 016 01 - Diskussion Zuletzt bearbeitet: 2016-05-21 18:26:38 Schumann | Br, Fragment, Gesichtet, Golding et al 1999, KomplettPlagiat, SMWFragment, Schutzlevel sysop |
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Untersuchte Arbeit: Seite: 16, Zeilen: 1-3 |
Quelle: Golding et al 1999 Seite(n): 8797, Zeilen: li.Sp. 16-22 |
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[Calcium spikes could thus serve as a robust cellular mechanism by which synaptic inputs conveying temporally correlated information] might be selectively reinforced. This mechanism would be expected to function effectively in distal dendritic regions in which the influence of back propagating action potentials is comparatively weak (Spruston et al., 1995; Kamondi et al., 1998).
• Kamondi A, Acsády L, Buzsáki G (1998) Dendritic spikes are enhanced by cooperative network activity in the intact hippocampus. J Neurosci 18:3919-28. • Spruston N, Schiller Y, Stuart G, Sakmann B (1995) Activity-dependent action potential invasion and calcium influx into hippocampal CA1 dendrites. Science 268:297-300. |
Calcium spikes could thus serve as a robust cellular mechanism by which synaptic inputs conveying temporally correlated information might be selectively reinforced. This mechanism would be expected to function effectively in distal dendritic regions in which the influence of backpropagating action potentials is comparatively weak (Spruston et al., 1995; Kamondi et al., 1998).
Kamondi A, Acsady L, Buzsa´ki G (1998) Dendritic spikes are enhanced by cooperative network activity in the intact hippocampus. J Neurosci 18:3919–3928. Spruston N, Schiller Y, Stuart G, Sakmann B (1995) Activity-dependent action potential invasion and calcium influx into hippocampal CA1 dendrites. Science 268:297–300. |
Ohne Hinweis auf eine Übernahme. |
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Letzte Bearbeitung dieser Seite: durch Benutzer:Schumann, Zeitstempel: 20160521182721