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Arteriogenesis in Gja5 (Connexin-40) deficient mice

von Dr. Haitao Wang

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[1.] Haw/Fragment 013 01 - Diskussion
Zuletzt bearbeitet: 2014-10-23 19:33:47 WiseWoman
BauernOpfer, Buschmann and Schaper 1999, Fragment, Gesichtet, Haw, SMWFragment, Schutzlevel sysop

Typus
BauernOpfer
Bearbeiter
Hindemith
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Untersuchte Arbeit:
Seite: 13, Zeilen: 1-9
Quelle: Buschmann and Schaper 1999
Seite(n): 124, Zeilen: l. Spalte: 43ff
[In another study, Arras, M., et al.[14] injected a single dose of] lipopolysaccharide intravenously into New Zealand white rabbits 3 days after ligation of the femoral artery. This potent stimulator of TNF-a also markedly enhanced the number of monocyte-derived macrophages accumulated around growing collateral arteries. Peripheral and collateral conductance was markedly increased. Nevertheless, on a molar basis MCP-1 is the most potent arteriogenic peptide[21]. VEGF is a peptide with angiogenic properties. It is produced by cells in close vicinity of endothelial cells, its chemoattractive action on monocytes is dose dependent; and its expression is highly regulated by hypoxia and hence by a physiological feedback mechanism to tissue hypoxia[67].

14. Arras, M., et al., Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb. J Clin Invest, 1998. 101(1): p. 40-50.

21. Buschmann, I. and W. Schaper, Arteriogenesis Versus Angiogenesis: Two Mechanisms of Vessel Growth. News Physiol Sci, 1999. 14: p. 121-125.

67. Carmeliet, P. and D. Collen, Vascular development and disorders: molecular analysis and pathogenic insights. Kidney Int, 1998. 53(6): p. 1519-49.

In another study, we injected a single dose of lipopolysaccharide intravenously into New Zealand White rabbits 3 days after ligation of the femoral artery (1). This potent stimulator of tumor necrosis factor- a also markedly enhanced the number of monocyte-derived macrophages accumulated around growing collateral arteries. Peripheral and collateral conductances were markedly increased. Nevertheless, on a molar basis MCP-1 is the most potent arteriogenic peptide. Vascular endothelial growth factor (VEGF) is a peptide with angiogenic properties. It is produced by cells in close vicinity of endothelial cells, suggesting paracrine regulation of capillary formation; it is secreted and exerts a direct effect via interaction with endothelial receptors Flk-1 and Flt-1; its chemoattractive action on monocytes is dose dependent; and its expression is highly regulated by hypoxia and thereby a physiological feedback mechanism to tissue hypoxia (3).

1. Arras, M., W. D. Ito, D. Scholz, B. Winkler, J. Schaper, and W. Schaper. Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb. J. Clin. Invest. 101: 40–50, 1997.

3. Carmeliet, P., and D. Collen. Vascular development and disorders—molecular analysis and pathogenic insights. Kidney Int. 53: 1519–1549, 1998.

Anmerkungen

Die Quelle ist in der Mitte genannt, der Umfang der Übernahme wird durch diesen Verweis aber nicht klar. Auch zwei Literaturverweise sind übernommen.

Sichter
(Hindemith), WiseWoman

[2.] Haw/Fragment 013 15 - Diskussion
Zuletzt bearbeitet: 2014-10-15 19:07:03 Singulus
Fragment, Gesichtet, Haw, KomplettPlagiat, Limbourg et al 2009, SMWFragment, Schutzlevel sysop

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Untersuchte Arbeit:
Seite: 13, Zeilen: 15-18
Quelle: Limbourg et al 2009
Seite(n): 1737, Zeilen: l. Spalte: 15ff
Angiogenesis, i.e., the sprouting of capillaries from the pre-existing vasculature, is mainly initiated by hypoxia in ischemic tissue[3]. These newly formed capillaries consist of endothelial tubes lacking proper wall structures. Angiogenesis alone has a limited capacity to increase perfusion of the surrounding ischemic tissue.

3. Carmeliet, P., Mechanisms of angiogenesis and arteriogenesis. Nat Med, 2000. 6(4): p. 389-95.

Angiogenesis, i.e., the sprouting of capillaries from the pre-existing vasculature, is mainly initiated by hypoxia in ischemic tissue4. These newly formed capillaries consist of endothelial tubes lacking proper wall structures. Angiogenesis alone has a limited capacity to increase perfusion of the surrounding ischemic tissue.

4. Carmeliet, P. Mechanisms of angiogenesis and arteriogenesis. Nat. Med. 6, 389–395 (2000).

Anmerkungen

Ein Verweis auf die Quelle fehlt.

Sichter
(Hindemith) Klgn

[3.] Haw/Fragment 013 20 - Diskussion
Zuletzt bearbeitet: 2014-10-20 22:11:24 WiseWoman
BauernOpfer, Buschmann and Schaper 1999, Fragment, Gesichtet, Haw, SMWFragment, Schutzlevel sysop

Typus
BauernOpfer
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 13, Zeilen: 20-29
Quelle: Buschmann and Schaper 1999
Seite(n): 121, 122, Zeilen: 121: r. Spalte: letzte Zeilen; 122: l. Spalte: 1ff
Angiogenesis is a process by which new capillary blood vessels sprout from a pre-existing blood vessel[68]. It is an important component of various normal and pathological conditions such as wound healing, fracture repair, folliculogenesis, ovulation, and pregnancy. These periods of angiogenesis are tightly regulated. However, if not properly controlled, angiogenesis can also represent a significant pathogenic component of tumor growth and metastasis, rheumatic arthritis, and retinopathies[21]. Angiogenesis is a complex phenomenon consisting of several distinct processes, which include endothelial migration and proliferation, extracellular proteolysis, endothelial differentiation (capillary tube formation), and vascular wall remodeling. It is important to recognize that these newly formed capillary tubes lack vascular smooth muscle cells.

21. Buschmann, I. and W. Schaper, Arteriogenesis Versus Angiogenesis: Two Mechanisms of Vessel Growth. News Physiol Sci, 1999. 14: p. 121-125.

68. Risau, W., Mechanisms of angiogenesis. Nature, 1997. 386(6626): p. 671-4.

Angiogenesis is a process by which new capillary blood vessels sprout from a preexisting blood vessel

[Seite 122]

(10). It is an important component of various normal and pathological conditions such as wound healing, fracture repair, folliculogenesis, ovulation, and pregnancy. These periods of angiogenesis are tightly regulated. However, if not properly controlled, angiogenesis can also represent a significant pathogenic component of tumor growth and metastasis, rheumatic arthritis, and retinopathies. Angiogenesis is a complex phenomenon consisting of several distinct processes, which include endothelial migration and proliferation, extracellular proteolysis, endothelial differentiation (capillary tube formation), and vascular wall remodeling. It is important to recognize that these newly formed capillary tubes lack vascular smooth muscle cells.


10. Risau, W. Mechanisms of angiogenesis. Nature 386: 671–674, 1997.

Anmerkungen

Auf die Quelle wird in der Mitte der Passage verwiesen, der Umfang der Übernahme bleibt so im Unklaren.

Sichter
(Hindemith) Klgn


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