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Typus
BauernOpfer
Bearbeiter
Hindemith
Gesichtet
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Untersuchte Arbeit:
Seite: 13, Zeilen: 1-9
Quelle: Buschmann and Schaper 1999
Seite(n): 124, Zeilen: l. Spalte: 43ff
[In another study, Arras, M., et al.[14] injected a single dose of] lipopolysaccharide intravenously into New Zealand white rabbits 3 days after ligation of the femoral artery. This potent stimulator of TNF-a also markedly enhanced the number of monocyte-derived macrophages accumulated around growing collateral arteries. Peripheral and collateral conductance was markedly increased. Nevertheless, on a molar basis MCP-1 is the most potent arteriogenic peptide[21]. VEGF is a peptide with angiogenic properties. It is produced by cells in close vicinity of endothelial cells, its chemoattractive action on monocytes is dose dependent; and its expression is highly regulated by hypoxia and hence by a physiological feedback mechanism to tissue hypoxia[67].

14. Arras, M., et al., Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb. J Clin Invest, 1998. 101(1): p. 40-50.

21. Buschmann, I. and W. Schaper, Arteriogenesis Versus Angiogenesis: Two Mechanisms of Vessel Growth. News Physiol Sci, 1999. 14: p. 121-125.

67. Carmeliet, P. and D. Collen, Vascular development and disorders: molecular analysis and pathogenic insights. Kidney Int, 1998. 53(6): p. 1519-49.

In another study, we injected a single dose of lipopolysaccharide intravenously into New Zealand White rabbits 3 days after ligation of the femoral artery (1). This potent stimulator of tumor necrosis factor- a also markedly enhanced the number of monocyte-derived macrophages accumulated around growing collateral arteries. Peripheral and collateral conductances were markedly increased. Nevertheless, on a molar basis MCP-1 is the most potent arteriogenic peptide. Vascular endothelial growth factor (VEGF) is a peptide with angiogenic properties. It is produced by cells in close vicinity of endothelial cells, suggesting paracrine regulation of capillary formation; it is secreted and exerts a direct effect via interaction with endothelial receptors Flk-1 and Flt-1; its chemoattractive action on monocytes is dose dependent; and its expression is highly regulated by hypoxia and thereby a physiological feedback mechanism to tissue hypoxia (3).

1. Arras, M., W. D. Ito, D. Scholz, B. Winkler, J. Schaper, and W. Schaper. Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb. J. Clin. Invest. 101: 40–50, 1997.

3. Carmeliet, P., and D. Collen. Vascular development and disorders—molecular analysis and pathogenic insights. Kidney Int. 53: 1519–1549, 1998.

Anmerkungen

Die Quelle ist in der Mitte genannt, der Umfang der Übernahme wird durch diesen Verweis aber nicht klar. Auch zwei Literaturverweise sind übernommen.

Sichter
(Hindemith), WiseWoman

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