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[1.] Iam/Fragment 004 01 - Diskussion
Zuletzt bearbeitet: 2014-03-13 12:23:13 Graf Isolan
Fragment, Gesichtet, Iam, KomplettPlagiat, Quesaraku et al. 2009, SMWFragment, Schutzlevel sysop

Typus
KomplettPlagiat
Bearbeiter
Singulus
Gesichtet
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Untersuchte Arbeit:
Seite: 4, Zeilen: 1-2
Quelle: Quesaraku et al. 2009
Seite(n): 910, Zeilen: r. col. 10-15
[However, beyond this, there are further] mechanisms involved in the actions occurring after the exposure of cells and tissue to ionizing radiation, such as radiation-induced apoptosis (Hasegawa et al. 2002).

Hasegawa M, Imai R, Nojima K, Sakurai H, Suzuki Y, Kawashima M, Matsuura M, Nakamura Y, Nakano T (2002) [Radiation-induced apoptosis in vivo: therapeutic significance of apoptosis in radiation therapy]. Nippon Igaku Hoshasen Gakkai Zasshi 62:535-539

However, beyond this, there are further mechanisms involved in the actions occurring after the exposure of cells and tissue to ionizing radiation, such as radiation-induced apoptosis (25) and radiation-induced changes in gene expression (26–28).

25. Hasegawa M, Imai R, Nojima K, et al. Radiation-induced apoptosis in vivo: therapeutic significance of apoptosis in radiation therapy. Nippon Igaku Hoshasen Gakkai Zasshi 2002; 62: 535–9.

26. Christiansen H, Batusic D, Saile B, et al. Identification of genes responsive to gamma radiation in rat hepatocytes and rat liver by cDNA array gene expression analysis. Radiat Res 2006; 165: 318–25.

27. Christiansen H, Sheikh N, Saile B, et al. X-irradiation in rat liver: consequent upregulation of hepcidin and downregulation of hemojuvelin and ferroportin-1 gene expression. Radiology 2007; 242: 189–97.

28. Moriconi F, Christiansen H, Raddatz D, et al. Effect of radiation on gene expression of rat liver chemokines: in vivo and in vitro studies. Radiat Res 2008; 169: 162–9.

Anmerkungen

Nothing has been marked as a citation.

Sichter
(Singulus), Graf Isolan

[2.] Iam/Fragment 004 03 - Diskussion
Zuletzt bearbeitet: 2014-03-13 22:00:51 Singulus
Fragment, Gesichtet, Iam, KomplettPlagiat, Moriconi et al 2008, SMWFragment, Schutzlevel sysop

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Graf Isolan
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Untersuchte Arbeit:
Seite: 4, Zeilen: 3-10
Quelle: Moriconi et al 2008
Seite(n): 162, Zeilen: left col. 29-35 - right col. 1-2.12-15
The interest of gastroenterologists, radiation oncologists and pathologists in radiation-induced liver disease has been generated by clinical observations of the deterioration of liver function occurring after abdominal irradiation with at least 30 Gy in patients with chronic liver diseases or even in patients with a healthy liver. This toxicity limits the use of radioimmunotherapy for malignant lymphomas, radio-chemotherapy for biliopancreatic carcinomas, and radiotherapy for liver malignancies (Greco et al. 2004;Wang et al. 1995). The molecular pathogenesis of hepatocellular damage after irradiation and the development of radiation-induced liver disease is still unknown.

Greco C, Catalano G, Di GA, Orecchia R (2004) Radiotherapy of liver malignancies. From whole liver irradiation to stereotactic hypofractionated radiotherapy. Tumori 90:73-79

Wang S, Quadri SM, Tang XZ, Stephens LC, Lollo CP, Bartholomew RM, Vriesendorp HM (1995) Liver toxicity induced by combined external-beam irradiation and radioimmunoglobulin therapy. Radiat Res 141:294-302

Introduction

The interest of gastroenterologists, radiation oncologists and pathologists in radiation-induced liver disease has been generated by clinical observations of the deterioration of liver function occurring after abdominal irradiation with at least 30 Gy in patients with chronic liver diseases or even in patients with a healthy liver. This toxicity limits the use of radioimmunotherapy for malignant lymphomas, radio-chemotherapy for biliopancreatic carcinomas, and radiotherapy for liver malignancies (1-3).

[...] The molecular pathogenesis of hepatocellular damage after irradiation and the development of radiation-induced liver disease is still unknown.


1. S. Wang, S. M. Quadri, X. Z. Tang, L. C. Stephens, C. T. Lollo, R. M. Bartholomew and H. M. Vriesendorp, Liver toxicity induced by combined external-beam irradiation and radioimmunoglobulin therapy. Radiat. Res. 141, 294–302 (1995).

2. C. Sempoux, Y. Horsmans, A. Geubel, J. Fraikin, B. E. Van Beers, J. F. Gigot, J. Lerut and J. Rahier, Severe radiation-induced liver disease following localized radiation therapy for biliopancreatic carcinoma: activation of hepatic stellate cells as an early event. Hepatology 26, 128–134 (1997).

3. C. Greco, G. Catalano, A. Di Grazia and R. Orecchia, Radiotherapy of liver malignancies. From whole liver irradiation to stereotactic hypofractionated radiotherapy. Tumori 90, 73–79 (2004).

Anmerkungen

No part of this has been marked as a citation.

Sichter
(Graf Isolan) Schumann

[3.] Iam/Fragment 004 12 - Diskussion
Zuletzt bearbeitet: 2014-04-06 12:13:24 Guckar
Butterfield et al 2006, Fragment, Gesichtet, Iam, KomplettPlagiat, SMWFragment, Schutzlevel sysop

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Hindemith
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Untersuchte Arbeit:
Seite: 4, Zeilen: 12-17
Quelle: Butterfield et al 2006
Seite(n): 457, Zeilen: l.col: 19-23, 32-36
Inflammation is often used to describe a series of signs and symptoms after soft tissue or bone injury. Appropriately, this term was originally used to describe the 4 classic signs of the affected tissue’s response to trauma: redness, swelling, heat and pain (Rocha e Silva 1978). The inflammatory process can be initiated through a variety of mechanisms, which include the introduction of pathogens as well as challenges to the system through chemical, thermal and mechanical stresses. Regardless of the inciting factors, the events accompanying [inflammation are somewhat consistent.]

Rocha e Silva (1978) A brief survey of the history of inflammation. Agents Actions 8:45-49

In sports medicine, the term inflammation is often used to describe a series of signs and symptoms after soft tissue or bony injury. Appropriately, this term was originally used to describe the 4 classic signs of the affected tissue’s response to trauma: redness, swelling, heat, and pain.1 [...]

The inflammatory process can be initiated through a variety of mechanisms, which include the introduction of pathogens as well as challenges to the system through chemical, thermal, and mechanical stresses. Regardless of the inciting factors, the events accompanying inflammation are somewhat consistent.


1. Rocha e Silva M. A brief survey of the history of inflammation, 1978. Agents Actions. 1994;43:86–90.

Anmerkungen

The source is not mentioned here.

Sichter
(Hindemith) Schumann


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