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[1.] Iam/Fragment 006 01 - Diskussion
Zuletzt bearbeitet: 2014-04-06 12:13:41 Guckar
BauernOpfer, Butterfield et al 2006, Fragment, Gesichtet, Iam, SMWFragment, Schutzlevel sysop

Typus
BauernOpfer
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 6, Zeilen: 1-3
Quelle: Butterfield et al 2006
Seite(n): 458, Zeilen: l.col: 15-19
[An early] intervention may prove the most beneficial strategy to minimizing tissue injury and facilitating tissue repair and recovery of function after soft bone injury (Butterfield et al. 2006). Although the implications for clinical therapeutics are not fully realized at this time, early intervention may prove the most beneficial strategy to minimizing tissue injury and facilitating tissue repair and recovery of function.
Anmerkungen

The copied text starts already on the previous page: Iam/Fragment 005 08

Although the source is given, it is not clear to the reader how much text has been taken and that the text has been copied verbatim.

Sichter
(Hindemith) Schumann

[2.] Iam/Fragment 006 20 - Diskussion
Zuletzt bearbeitet: 2014-03-12 19:44:25 Graf Isolan
Fragment, Gesichtet, Iam, SMWFragment, Schutzlevel sysop, Sheikh 2006, Verschleierung

Typus
Verschleierung
Bearbeiter
Graf Isolan
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 6, Zeilen: 20-28
Quelle: Sheikh 2006
Seite(n): 4, Zeilen: 8-16
Interleukin (IL)-6 itself from the group of IL-6-type cytokines and IL-1β together with tumor necrosis factor (TNF)-α from the IL-1-type cytokine group are considered to be the major mediators of the acute-inflamamtion. At the inflammatory sites, IL-6 is produced by macrophages, endothelial cells and fibroblasts (Ramadori and Christ 1999). The release of mature IL-1β by macrophages seems to take place only during or after cell death (Perregaux and Gabel 1998). TNF-α is synthesized mainly by mononuclear phagocytes recruited at the sites of damage and by tissue macrophages. While IL-6, IL-1β, and TNF-α are the inducers of acute phase protein gene expression, other cytokines were shown to modulate this expression (Ramadori and Christ 1999).

Perregaux DG, Gabel CA (1998) Post-translational processing of murine IL-1: evidence that ATP-induced release of IL-1 alpha and IL-1 beta occurs via a similar mechanism. J Immunol 160:2469-2477

Ramadori G, Christ B (1999) Cytokines and the hepatic acute-phase response. Semin Liver Dis 19:141-155

Interleukin- (IL) 6 from the group of IL-6-type cytokines and IL-1β together with tumor necrosis factor (TNF)-α from the group of IL-1-type cytokine are considered to be the major mediators of the APR. At the inflammatory sites, IL-6 is produced by macrophages, endothelial cells, and fibroblasts (Ramadori and Christ, 1999). The release of mature IL-1β by macrophages seems to take place only during or after cell death (Perregaux and Gabel, 1998). TNF-α is synthesized mainly by mononuclear phagocytes recruited at the sites of damage and by tissue macrophages (Ramadori and Christ, 1999). While IL-6, IL-1β, and TNF-α are the inducers of acute-phase protein gene expression, other cytokines (Table 1) were shown to modulate this expression (Moshage, 1997).

Moshage H. Cytokines and the hepatic acute phase response. J Pathol 181: 257-266, 1997.

Perregaux DG and Gabel CA. Post-translational processing of murine IL-1: evidence that ATP-induced release of IL-1{alpha} and IL-1{beta} occurs via a similar mechanism. J Immunol 160: 2469-2477, 1998.

Ramadori G and Christ B. Cytokines and the hepatic acute-phase response. Semin Liver Dis 19: 141-155, 1999.

Anmerkungen

Nearly identical - nevertheless nothing has been marked as a citation.

Sichter
(Graf Isolan) Schumann


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