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Reconsolidation: Behavioural and Electrophysiological Sequelae of Context and Stress in Human Episodic Memory

von Dr. Jennifer L. Moore

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[1.] Jm/Fragment 070 01 - Diskussion
Zuletzt bearbeitet: 2014-01-12 21:16:52 Graf Isolan
Fragment, Gesichtet, Jm, KomplettPlagiat, SMWFragment, Scanlon et al 2006, Schutzlevel sysop

Typus
KomplettPlagiat
Bearbeiter
Agrippina1
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 70, Zeilen: 1-14
Quelle: Scanlon et al 2006
Seite(n): 5, Zeilen: 3rd col: 37ff
It has been repeatedly demonstrated by correlating scalp-recorded EEG with intracranial neuronal discharges in the monkey and the cat, that the polarity of ERPs, are related to either excitation or inhibition of cells. Comparison of evoked potentials and neuronal spiking activity reveals that neuronal discharges/firing in thalamocortical cells seem to result in negative ERP components, while cellular inhibition underlies positive potentials. Thus EPSPs/depolarisations appear responsible for negative ERP deflections, while IPSPs/hyperpolarisations are the cause of scalp-recorded positivities. Specifically, the scalp recorded negative shifts seem to be due to the depolarisation of pyramidal cell dendrites, which results in an extracellular surface current sink, with the opposite situation the case for scalp recorded positives. The relationship between neuronal activity and scalp-recorded potentials is shown in Figures 2.5 and 2.6 above, from Coenen (1995). Although this polarity reversal between intracranial and scalp recorded activity is true in most cases, the opposite relationship, where scalp positives are due to neuronal excitation and negatives to inhibition, has also been found on occasion. It has been repeatedly demonstrated by correlating scalp recorded EEG with intracranial neuronal discharges in the monkey and the cat that the polarity of ERPs are related to either excitation or inhibition of cells. Comparison of evoked potentials and neuronal spiking activity reveals that neuronal discharges/firing in thalamocortical cells seem to result in negative ERP components, while cellular inhibition underlies positive potentials. Thus EPSPs/depolarisations appear responsible for negative ERP deflections, while IPSPs/hyperpolarisations are the cause of scalp-recorded positivities. Specifically, the scalp recorded negative shifts seem to be due to the depolarisation of pyramidal cell dendrites, which results in an extracellular surface current sink, with the opposite situation the case for scalp recorded positives. The relationship between neuronal activity and scalp-recorded potentials is shown in Figures 3 and 4, from Coenen (1995). Although this polarity reversal between intracranial and scalp recorded activity is true in most cases, the opposite relationship, where scalp positives are due to neuronal excitation and negatives to inhibition, has also been found on occasion.
Anmerkungen

A reference to the source is missing.

Sichter
Hindemith

[2.] Jm/Fragment 070 22 - Diskussion
Zuletzt bearbeitet: 2014-01-12 21:16:57 Graf Isolan
Fragment, Gesichtet, Jm, SMWFragment, Scanlon et al 2006, Schutzlevel sysop, Verschleierung

Typus
Verschleierung
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 70, Zeilen: 22-24
Quelle: Scanlon et al 2006
Seite(n): 5, Zeilen: 3rd. col: last paragraph
Given that electrical potentials travel through both the bone and skin of the skull and scalp at high speed leading to almost instantaneous recording of the electrical activity of the brain, ERPs provide very [high temporal resolution.] Electrical potentials travel through the bone and skin of the skull and scalp at high speed leading to almost instantaneous recording of the electrical activity of the brain, thus providing very high temporal resolution.
Anmerkungen

A reference to the source is missing. The copied text continues on the next page: Jm/Fragment_071_01

Sichter
(Hindemith) Agrippina1


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