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| Untersuchte Arbeit: Seite: 29, Zeilen: 17-25 |
Quelle: Smeets et al 2007 Seite(n): 116, Zeilen: l.col: 1ff |
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| Exposure to highly stressful events is known to trigger a variety of physiological reactions, of which many are related to the activation of stress-responsive sympatho adrenal-medullary (SAM) and hypothalamic–pituitary–adrenal (HPA) axes. A plethora of research has revealed that secretion of glucocorticoids (GCs) in response to HPA axis stimulation may modulate memory functioning (e.g., de Kloet et al., 1999; McGaugh, 2000; Roozendaal, 2000). However, the precise direction of stress-induced GC effects on memory performance is far from succinct. Animal studies, for example, have shown that GCs can exert both facilitating (e.g., on aversive conditioning) as well as impairing effects on memory (e.g., de Kloet et al., 1999; Lupien & McEwen, 1997; McGaugh & Roozendaal, 2002). | Most people are familiar with highly stressful events. Exposure to such events is known to trigger a variety of physiological reactions, of which many are related to the activation of stress-responsive sympathoadrenal medullary (SAM) and hypothalamic–pituitary–adrenal (HPA) axes. A plethora of research has revealed that secretion of glucocorticoids (GCs) due to HPA axis stimulation may modulate memory functioning (e.g., de Kloet et al., 1999; McGaugh, 2000; Roozendaal, 2000). However, the precise direction of stress-induced GC effects on memory performance is far from clear. Animal studies, for example, have shown that GCs can have facilitating (e.g., on aversive conditioning), but also impairing effects on memory (e.g., de Kloet et al., 1999; Lupien and McEwen, 1997; McGaugh and Roozendaal, 2002). |
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