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Quelle:Arc/Ushio-Fukai 2006

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Angaben zur Quelle [Bearbeiten]

Autor     Masuko Ushio-Fukai
Titel    Redox signaling in angiogenesis: Role of NADPH oxidase
Zeitschrift    Cardiovascular Research
Herausgeber    European Society of Cardiology
Verlag    Elsevier
Ausgabe    71
Jahr    2006
Seiten    226 – 235
DOI    10.1016/j.cardiores.2006.04.015
URL    http://cardiovascres.oxfordjournals.org/content/71/2/226.full.pdf

Literaturverz.   

no
Fußnoten    yes
Fragmente    1


Fragmente der Quelle:
[1.] Arc/Fragment 037 01 - Diskussion
Zuletzt bearbeitet: 2014-03-06 09:16:13 Singulus
Arc, BauernOpfer, Fragment, Gesichtet, SMWFragment, Schutzlevel sysop, Ushio-Fukai 2006

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Untersuchte Arbeit:
Seite: 37, Zeilen: 0-1
Quelle: Ushio-Fukai 2006
Seite(n): 231, Zeilen: 1ff
[In Fig. 10, Ushio-Fukai describes important ROS-dependent genes and proteins associated with anigiogenesis, like urokinase plasminogen activator (uPA), plasminogen activator inhibitor-1 (PAI-1) and metalloproteinases (MMPs).]

37a diss.png

Fig. 10 - Role of ROS derived from NADPH oxidase in the induction of transcription factors and genes involved in angiogenesis. Ischemia/hypoxia stimulates VEGF induction and NADPH oxidase, thereby promoting activation of redox signalling events and induction of redox sensitive transcription factors and genes involved in angiogenesis. ROS derived from NADPH oxidase also upregulate mitochondrial SOD (MnSOD), increasing mitochondrial H2O2 production, which could represent a feed-forward mechanism by which ROS-triggered H2O2 formation plays an important role in angiogenesis101.


101. Tuttle JL, Sanders BM, Burkhart HM, Fath SW, Kerr KA, Watson WC, Herring BP, Dalsing MC, Unthank JL. Impaired collateral artery development in spontaneously hypertensive rats. Microcirculation. 2002;9:343-351.

37a source.png

Fig. 4. Role of ROS derived from NADPH oxidase in induction of transcription factors and genes involved in angiogenesis. Ischemia/hypoxia stimulates VEGF induction and NADPH oxidase, thereby promoting activation of redox signaling events and induction of redox sensitive transcription factors and genes involved in angiogenesis. ROS derived from NADPH oxidase also upregulate MnSOD which increases mitochondrial H2O2 production, which could represent a feed-forward mechanism by which ROS-triggered H2O2 formation plays an important role in angiogenesis.

Anmerkungen

On the previous page it is mentioned that "In Fig. 10, Ushio-Fukai describes important ...". However, there is no reference to a publication by Usio-Fukai to be found in the list of references, and it is also not clear that the caption of figure 10 is also taken from that source.

In the given source Tuttle et al. (2002) none of the copied material can be found.

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