Fandom

VroniPlag Wiki

Quelle:Iam/Butterfield et al 2006

< Quelle:Iam

31.371Seiten in
diesem Wiki
Seite hinzufügen
Diskussion0

Störung durch Adblocker erkannt!


Wikia ist eine gebührenfreie Seite, die sich durch Werbung finanziert. Benutzer, die Adblocker einsetzen, haben eine modifizierte Ansicht der Seite.

Wikia ist nicht verfügbar, wenn du weitere Modifikationen in dem Adblocker-Programm gemacht hast. Wenn du sie entfernst, dann wird die Seite ohne Probleme geladen.

Angaben zur Quelle [Bearbeiten]

Autor     Timothy A. Butterfield, Thomas M. Best, Mark A. Merrick
Titel    The Dual Roles of Neutrophils and Macrophages in Inflammation: A Critical Balance Between Tissue Damage and Repair
Zeitschrift    Journal of Athletic Training
Herausgeber    National Athletic Trainers’ Association
Ausgabe    41
Jahr    2006
Nummer    4
Seiten    457-465
URL    http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1748424/pdf/i1062-6050-41-4-457.pdf

Literaturverz.   

yes
Fußnoten    yes
Fragmente    5


Fragmente der Quelle:
[1.] Iam/Fragment 001 05 - Diskussion
Zuletzt bearbeitet: 2014-03-13 21:45:50 Singulus
Butterfield et al 2006, Fragment, Iam, KeineWertung, SMWFragment, Schutzlevel, ZuSichten

Typus
KeineWertung
Bearbeiter
Singulus
Gesichtet
No.png
Untersuchte Arbeit:
Seite: 5, Zeilen: 1
Quelle: Butterfield et al 2006
Seite(n): 475, Zeilen: l.col: 35-36
[Regardless of the inciting factors, the events accompanying] inflammation are somewhat consistent. Regardless of the inciting factors, the events accompanying inflammation are somewhat consistent.
Anmerkungen
Sichter
(Singulus)

[2.] Iam/Fragment 004 12 - Diskussion
Zuletzt bearbeitet: 2014-04-06 12:13:24 Guckar
Butterfield et al 2006, Fragment, Gesichtet, Iam, KomplettPlagiat, SMWFragment, Schutzlevel sysop

Typus
KomplettPlagiat
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 4, Zeilen: 12-17
Quelle: Butterfield et al 2006
Seite(n): 457, Zeilen: l.col: 19-23, 32-36
Inflammation is often used to describe a series of signs and symptoms after soft tissue or bone injury. Appropriately, this term was originally used to describe the 4 classic signs of the affected tissue’s response to trauma: redness, swelling, heat and pain (Rocha e Silva 1978). The inflammatory process can be initiated through a variety of mechanisms, which include the introduction of pathogens as well as challenges to the system through chemical, thermal and mechanical stresses. Regardless of the inciting factors, the events accompanying [inflammation are somewhat consistent.]

Rocha e Silva (1978) A brief survey of the history of inflammation. Agents Actions 8:45-49

In sports medicine, the term inflammation is often used to describe a series of signs and symptoms after soft tissue or bony injury. Appropriately, this term was originally used to describe the 4 classic signs of the affected tissue’s response to trauma: redness, swelling, heat, and pain.1 [...]

The inflammatory process can be initiated through a variety of mechanisms, which include the introduction of pathogens as well as challenges to the system through chemical, thermal, and mechanical stresses. Regardless of the inciting factors, the events accompanying inflammation are somewhat consistent.


1. Rocha e Silva M. A brief survey of the history of inflammation, 1978. Agents Actions. 1994;43:86–90.

Anmerkungen

The source is not mentioned here.

Sichter
(Hindemith) Schumann

[3.] Iam/Fragment 005 08 - Diskussion
Zuletzt bearbeitet: 2014-04-06 12:12:36 Guckar
BauernOpfer, Butterfield et al 2006, Fragment, Gesichtet, Iam, SMWFragment, Schutzlevel sysop

Typus
BauernOpfer
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 5, Zeilen: 8-19
Quelle: Butterfield et al 2006
Seite(n): 457, 458, 459, Zeilen: 457: r.col: 33-37; 458: l.col:15-19; 459: l.col: 12-15,22-27,33-34, r.col: 7-10
The cellular processes of inflammation are regulated by a series of specific cell signals that stimulate a variety of cell types, resulting in a cascade of events including white blood cell (WBC) recruitment and activation. Neutrophils are the first subpopulation of WBCs to enter traumatized or stressed tissues (Cannon and St Pierre 1998). The traditional thinking has been that these cytokines were released only by injured or stressed tissue, resulting in the localization of neutrophils to these injured tissues. The role of neutrophils in liver injury remains controversial. Although neutrophils contribute to liver damage by generating reactive oxygen species (ROS) (Jaeschke and Hasegawa 2006).

Currently, researchers are focused on the mechanisms for neutrophil recruitment and the function of these neutrophils in otherwise healthy, uninjured tissue. Among the most important questions regarding neutrophils and inflammation is whether the localization of neutrophils after passing the endothelium facilitates healing or tissue destruction. An early [intervention may prove the most beneficial strategy to minimizing tissue injury and facilitating tissue repair and recovery of function after soft bone injury (Butterfield et al. 2006).]


Butterfield TA, Best TM, Merrick MA (2006) The dual roles of neutrophils and macrophages in inflammation: a critical balance between tissue damage and repair. J Athl Train 41:457-465

Cannon JG, St Pierre BA (1998) Cytokines in exertion-induced skeletal muscle injury. Mol Cell Biochem 179:159-167

Jaeschke H, Hasegawa T (2006) Role of neutrophils in acute inflammatory liver injury. Liver Int 26:912-919

[page 457]

The cellular processes of inflammation are regulated by a series of specific cell signals that stimulate a variety of cell types, resulting in a cascade of events including white blood cell (WBC) recruitment and activation.

[page 458]

Although the implications for clinical therapeutics are not fully realized at this time, early intervention may prove the most beneficial strategy to minimizing tissue injury and facilitating tissue repair and recovery of function.

[page 459]

The traditional thinking has been that these cytokines were released only by injured or damaged myocytes, resulting in the localization of neutrophils to these injured tissues. [...] Currently, researchers are focused on the mechanisms for neutrophil recruitment and the function of these neutrophils in otherwise healthy, uninjured muscle. Among the most important questions regarding neutrophils and inflammation is whether the localization of neutrophils in the ECM facilitates healing or tissue destruction. [...]

[...]

Neutrophils are the first subpopulation of WBCs to enter traumatized or stressed tissues.17 [...] Currently, the role of neutrophils in muscle injury remains controversial. Although neutrophils contribute to preexisting muscle damage,47 evidence is mounting that they may provide the principal insult to the muscle membrane.39


17. Cannon JG, St Pierre BA. Cytokines in exertion-induced skeletal muscle injury. Mol Cell Biochem. 1998;179:159–167.

39. Pizza FX, Koh TJ, McGregor SJ, Brooks SV. Muscle inflammatory cells after passive stretches, isometric contractions, and lengthening contractions. J Appl Physiol. 2002;92:1873–1878.

47. Frenette J, St-Pierre M, Cote CH, Mylona E, Pizza FX. Muscle impairment occurs rapidly and precedes inflammatory cell accumulation after mechanical loading. Am J Physiol Regul Integr Comp Physiol. 2002;282: R351–R357.

Anmerkungen

The source is given at the very end, but without indication that most of the preceding two paragraphs has been taken verbatim from it.

Note that the source is largely about muscle injury, whereas the thesis discusses liver damage in rats. The copied text is adapted accordingly.

Sichter
(Hindemith) Schumann

[4.] Iam/Fragment 006 01 - Diskussion
Zuletzt bearbeitet: 2014-04-06 12:13:41 Guckar
BauernOpfer, Butterfield et al 2006, Fragment, Gesichtet, Iam, SMWFragment, Schutzlevel sysop

Typus
BauernOpfer
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 6, Zeilen: 1-3
Quelle: Butterfield et al 2006
Seite(n): 458, Zeilen: l.col: 15-19
[An early] intervention may prove the most beneficial strategy to minimizing tissue injury and facilitating tissue repair and recovery of function after soft bone injury (Butterfield et al. 2006). Although the implications for clinical therapeutics are not fully realized at this time, early intervention may prove the most beneficial strategy to minimizing tissue injury and facilitating tissue repair and recovery of function.
Anmerkungen

The copied text starts already on the previous page: Iam/Fragment 005 08

Although the source is given, it is not clear to the reader how much text has been taken and that the text has been copied verbatim.

Sichter
(Hindemith) Schumann

[5.] Iam/Fragment 010 12 - Diskussion
Zuletzt bearbeitet: 2014-04-06 12:12:51 Guckar
Butterfield et al 2006, Fragment, Gesichtet, Iam, SMWFragment, Schutzlevel sysop, Verschleierung

Typus
Verschleierung
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 10, Zeilen: 12-16
Quelle: Butterfield et al 2006
Seite(n): 457, Zeilen: l.col: 37ff.
For research purposes, a reproducible injury model of tissue inflammation and repair is required; one of such model is radiation induced liver injury (Christiansen et al. 2006). Specifically, it serves as an accepted paradigm to aid in the understanding of the body’s generalized inflammatory process after irradiation and, therefore, of a significant number of liver injuries.

Christiansen H, Batusic D, Saile B, Hermann RM, Dudas J, Rave-Frank M, Hess CF, Schmidberger H, Ramadori G (2006) Identification of genes responsive to gamma radiation in rat hepatocytes and rat liver by cDNA array gene expression analysis. Radiat Res 165:318-325

For research purposes, a reproducible injury model of tissue inflammation and repair is required; one such model involves eccentric overload to skeletal muscle.2–4 [...] Specifically, it serves as an accepted paradigm to aid in the understanding of the body’s generalized inflammatory process and, therefore, of a significant number of athletic injuries.

2. Best TM, Fiebig R, Corr DT, Brickson S, Ji L. Free radical activity, antioxidant enzyme, and glutathione changes with muscle stretch injury in rabbits. J Appl Physiol. 1999;87:74–82.

3. Brickson S, Hollander J, Corr DT, Ji LL, Best TM. Oxidant production and immune response after stretch injury in skeletal muscle. Med Sci Sports Exerc. 2001;33:2010–2015.

4. Tsivitse SK, Mylona E, Peterson JM, Gunning WT, Pizza FX. Mechanical loading and injury induce human myotubes to release neutrophil chemoattractants. Am J Physiol Cell Physiol. 2005;288:C721–C729.

Anmerkungen

The source is not given, its text has been adapted from muscle to liver injuries.

Sichter
(Hindemith) Schumann

Auch bei Fandom

Zufälliges Wiki