Fandom

VroniPlag Wiki

Quelle:Mag/Boyton and Openshaw 2002

< Quelle:Mag

31.380Seiten in
diesem Wiki
Seite hinzufügen
Diskussion0

Störung durch Adblocker erkannt!


Wikia ist eine gebührenfreie Seite, die sich durch Werbung finanziert. Benutzer, die Adblocker einsetzen, haben eine modifizierte Ansicht der Seite.

Wikia ist nicht verfügbar, wenn du weitere Modifikationen in dem Adblocker-Programm gemacht hast. Wenn du sie entfernst, dann wird die Seite ohne Probleme geladen.

Angaben zur Quelle [Bearbeiten]

Autor     Rosemary J. Boyton, Peter J. Openshaw
Titel    Pulmonary defences to acute respiratory infection
Zeitschrift    British Medical Bulletin
Ausgabe    61
Jahr    2002
Seiten    1-12
URL    http://bmb.oxfordjournals.org/content/61/1/1.full.pdf

Literaturverz.   

yes
Fußnoten    yes
Fragmente    1


Fragmente der Quelle:
[1.] Mag/Fragment 086 05 - Diskussion
Zuletzt bearbeitet: 2014-03-16 17:02:38 Hindemith
BauernOpfer, Boyton and Openshaw 2002, Fragment, Gesichtet, Mag, SMWFragment, Schutzlevel sysop

Typus
BauernOpfer
Bearbeiter
Graf Isolan
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 86, Zeilen: 5-6, 8-14
Quelle: Boyton and Openshaw 2002
Seite(n): 1, Zeilen: 8-16 (abstract)
The respiratory tract is a fragile tissue with architecture that is finely designed for gas exchange. Due to this main function the lung is exposed to numerous pathogens and other harmful air pollutions and developed many mechanisms to prevent infectious and inflammations. In the first line of defence are structural mechanisms coming from barriers such as epithelial cell layers, mucus and cilia, which avoid the invasion of pathogens or antigens. A battery of mediators that constitute the innate response including lactoferin, lysozyme, collectins and defensins is followed. Activation of these molecules can lead directly to lysis of pathogens, or to destruction through opsonisation or the recruitment of inflammatory cells (Boyton et al., 2002).

Boyton RJ, Openshaw PJ. Pulmonary defences to acute respiratory infection. Br Med Bull. 2002; 61:1-12. Review.

The immune response to respiratory infection must, therefore, be rapid and efficient. However, the respiratory tract is a fragile tissue with architecture that is finely designed for gas exchange, so that the price of excessive or inappropriate inflammatory responses may itself be very high. The first line of defence comes from barriers such as mucus and cilia, followed by a battery of mediators that constitute the innate response. These include lactoferrin, lysozyme, collectins and defensins. Activation of these molecules can lead directly to lysis of pathogens, or to destruction through opsonisation or the recruitment of inflammatory cells.
Anmerkungen

Found in the chapter "Discussion" of the thesis. No part has been marked as a citation and for the reader it is not transparent to what extent the source has been used.

Sichter
(Graf Isolan), Hindemith

Auch bei Fandom

Zufälliges Wiki