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Angaben zur Quelle [Bearbeiten]

Autor     Daniel Gioeli, Ben E. Black, Vicki Gordon, Adam Spencer, Cristina T. Kesler, Scott T. Eblen,

Bryce M. Paschal, Michael J. Weber

Titel    Stress Kinase Signaling Regulates Androgen Receptor Phosphorylation, Transcription, and Localization
Zeitschrift    Molecular Endocrinology
Ausgabe    20
Jahr    2006
Nummer    3
Seiten    503-515
DOI    10.1210/me.2005-0351
URL    http://press.endocrine.org/doi/pdf/10.1210/me.2005-0351

Literaturverz.   

yes
Fußnoten    yes
Fragmente    2


Fragmente der Quelle:
[1.] Rlm/Fragment 047 01 - Diskussion
Zuletzt bearbeitet: 2014-12-01 17:27:18 Singulus
Fragment, Gesichtet, Gioeli et al 2006, KomplettPlagiat, Rlm, SMWFragment, Schutzlevel sysop

Typus
KomplettPlagiat
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 47, Zeilen: 1-7
Quelle: Gioeli et al 2006
Seite(n): 509, Zeilen: l.col: last paragraph
Discussion

The fundamental clinical problem for disseminated prostate cancer is the transition of androgen dependent desease [sic] to androgen independent desease [sic] after androgen ablation therapy. Collectively, data suggest that although advanced prostate cancer may be functionally independent of physiologic levels of androgen, it is not independent of the AR.

DISCUSSION

The fundamental clinical problem for disseminated prostate cancer is the transition of androgen-dependent disease to androgen-independent disease after androgen ablation therapy. Collectively, data suggest that although advanced prostate cancer may be functionally independent of physiologic levels of androgen, it is not independent of the AR (10, 12, 13, 23).


[...]

Anmerkungen

The source is not mentioned here.

Sichter
(Hindemith), SleepyHollow02

[2.] Rlm/Fragment 049 11 - Diskussion
Zuletzt bearbeitet: 2014-12-01 17:29:01 Singulus
BauernOpfer, Fragment, Gesichtet, Gioeli et al 2006, Rlm, SMWFragment, Schutzlevel sysop

Typus
BauernOpfer
Bearbeiter
Hindemith
Gesichtet
Yes.png
Untersuchte Arbeit:
Seite: 49, Zeilen: 11-20
Quelle: Gioeli et al 2006
Seite(n): 503, 504, Zeilen: 503: r.col: last line; 504: l.col: 1 ff.
Diverse agonists including activators of protein kinase A (forskolin) and protein kinase C [phorbol-12-myristate-13-acetate (PMA)] increased Ser 650 phosphorylation. Ser 650 phosphorylation occurs by both hormone-dependent and hormone- independent mechanisms (androgen, protein kinase A, EGF, and protein kinase C) suggest that modification of this site might be used to regulate steroid receptor function in response to a variety of physiological stimuli. Gioeli et al. have investigated which signal transduction pathways regulate Ser 650 phosphorylation and determined the effect of these signaling pathways on AR function. Notably, diverse agonists

[page 504]

including activators of protein kinase A (forskolin) and protein kinase C [phorbol-12-myristate-13-acetate (PMA)] increased Ser 650 phosphorylation (15). [...] The fact that Ser 650 phosphorylation occurs by both hormone-dependent and hormone-independent mechanisms (androgen, protein kinase A, EGF, and protein kinase C) suggest that modification of this site might be used to regulate steroid receptor function in response to a variety of physiological stimuli.

[...] In this study, we have investigated which signal transduction pathways regulate Ser 650 phosphorylation and determined the effect of these signaling pathways on AR function.

Anmerkungen

The source is mentioned in the text, but not in a way that suggests to the reader that even the text before is taken from it literally.

Sichter
(Hindemith), SleepyHollow02

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