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Angaben zur Quelle [Bearbeiten]

Autor     Meng-Lei Zhu and Natasha Kyprianou
Titel    Androgen receptor and growth factor signaling cross-talk in prostate cancer cells
Zeitschrift    Endocrine-Related Cancer
Ausgabe    15
Jahr    2008
Nummer    4
Seiten    841–849
DOI    10.1677/ERC-08-0084
URL    http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2740735/pdf/nihms124167.pdf

Literaturverz.   

ja
Fußnoten    ja
Fragmente    2


Fragmente der Quelle:
[1.] Shg/Fragment 013 14 - Diskussion
Zuletzt bearbeitet: 2014-10-29 18:44:29 Kybot
BauernOpfer, Fragment, SMWFragment, Schutzlevel, Shg, Zhu Kyprianou 2008, ZuSichten

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Untersuchte Arbeit:
Seite: 13, Zeilen: 14 ff.
Quelle: Zhu Kyprianou 2008
Seite(n): 841, Zeilen: abstract
The iAR activated target genes are an array of growth factor genes, e.g. epidermal growth factor (EGF); fibroblast growth factor (FGF); Insulin-like growth factor 1(IGF1); vascular endothelial growth factor (VEGF); transforming growth factor-β (TGFβ). The ability of iAR to cross-talk with key growth factor [signaling events toward the regulation of cell cycle, apoptosis, and differentiation outcomes in prostate cancer cells has been established.] The ability of AR to cross-talk with key growth factor signaling events toward the regulation of cell cycle, apoptosis, and differentiation outcomes in prostate cancer cells is established. In this paper, we review the functional interaction between AR and an array of growth factor signal transduction events (including epidermal growth factor; fibroblast growth factor; IGF1; vascular endothelial growth factor; transforming growth factor-b) in prostate tumors.

Androgen-induced prostate epithelial cell proliferation is regulated by an indirect pathway involving paracrine mediators produced by stromal cells, such as insulin-like growth factor (IGF), fibroblast growth factor (FGF), and epidermal growth factor (EGF; Cunha & Donjacour 1989, Byrne et al. 1996).

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[2.] Shg/Fragment 014 02 - Diskussion
Zuletzt bearbeitet: 2014-12-14 08:24:45 SleepyHollow02
BauernOpfer, Fragment, SMWFragment, Schutzlevel, Shg, Zhu Kyprianou 2008, ZuSichten

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Seite: 14, Zeilen: 2-11
Quelle: Zhu Kyprianou 2008
Seite(n): 843, Zeilen: figure 1
IGF, FGF, VEGF, and TGFβ secreted by the prostate stromal cells activate their receptors and interact with iAR signal axis. In prostate epithelial cells, the androgenic signal engages secreted VEGF and TGFβ which affect the prostate tumor microenvironment by inducing angiogenesis, stromal cell growth and differentiation. EGF signaling encounters iAR signal in a tight control of multiple pathways. Growth factor signaling may proceed via iAR signal and regulate the downstream effectors of iAR regulating key cellular processes including proliferation, differentiation, apoptosis, and survival of prostate cancer cells. [Meng-Lei Zhu, Natasha Kyprianou. 2008] IGF, FGF, VEGF, and TGFB secreted by the prostate stromal cells activate their receptors and interact with AR signal axis. In prostate epithelial cells, the androgenic signal engages secreted VEGF and TGFB which affects the prostate tumor microenvironment by inducing angiogenesis and stromal cell growth and differentiation. EGF signaling encounters AR signal in a tight control of multiple pathways. Growth factor signaling may proceed via AR signal and regulate the downstream effectors of AR regulating key cellular processes including proliferation, differentiation, apoptosis, and survival of prostate cancer cells.
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